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Has any research been done into the correlation of FnHL/HR and medial gastrocnemius muscle atrophy? If toe-off is happening with the foot in a more supinated position to compenste for the FnHL, is the medial muscle belly being used less than in a foot that has a normal ROM in the 1st mpj? Also with limited gastrocnemius extension due to FnHL how does one bulk up this muscle - and is a loss of muscle size of any notable biomechanical concern.
Has any research been done into the correlation of FnHL/HR and medial gastrocnemius muscle atrophy?
no....
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Has any research been done into the correlation of FnHL/HR and medial gastrocnemius muscle atrophy? If toe-off is happening with the foot in a more supinated position to compenste for the FnHL, is the medial muscle belly being used less than in a foot that has a normal ROM in the 1st mpj? Also with limited gastrocnemius extension due to FnHL how does one bulk up this muscle - and is a loss of muscle size of any notable biomechanical concern.
Any research in this area has to deal with the question of why FnHL sometimes has a more supinated toe off. By belief is that FnHL will cause pain if the gait is not altered and the patient subconsciously chooses a different gait pattern to avoid pain. So, supination muscles may be stronger in those patients who choose to have a more supinated toe off.
Is there any study that shows that the medial head of the gastroc produces significantly more supination than the lateral head of the gastroc?
FnHL - more questions than answers.
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